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On morphologic evaluation, the bone marrow is devoid of hematopoietic elements benadryl On morphologic evaluation, the bone marrow is dev benicar id of hematopoietic elements, showing largely fat cells. F bentyl ow cytometry shows that the CD34-cell population, which benzocaine ontains the stem cells and the early committed progen benzonatate tors, is substantially reduced. Data from in vitro colony-culture benzoyl peroxide assays suggest profound functional loss of the hem bextra topoietic progenitors, so much so that they are unresponsive ev biaxin n to high levels of hematopoietic growth factors. L biaxin xl ttle evidence points to a defective m
icroenviro bilberry ment as a cause of aplastic anemia. In patients biotin ith severe aplastic anemia (SAA), stromal cells have normal funct black cohosh on, including growth factor production. Adequate stromal functi blue n is implicit in the success of bone-marrow transpla boniva tation (BMT) in aplastic anemia because the st botox omal elements are frequently of host origin. The role of buprenorphine an immune dysfunction was suggested in 1970, when autologous r bupropion covery was documented in a patient with aplastic anem buspar a in whom engrafting failed after BMT. Mathe proposed that t buspirone e immunos
uppressive regimen used for conditioning butalbital romoted the return of normal marrow function. Since then, numero cpm s studies have shown that, in approximately 7 c2 % of patients with acquired aplastic anemia, immunosuppressive.